Pompe disease is an inherited disorder attributable to the buildup of a complex sugar called glycogen within the body's cells. The accumulation of glycogen in sure organs and tissues, particularly muscles, impairs their capacity to operate normally. Researchers have described three types of Pompe illness, which differ in severity and the age at which they appear. These types are referred to as traditional infantile-onset, non-traditional infantile-onset, and late-onset. The traditional type of infantile-onset Pompe disease begins inside a few months of beginning. Infants with this disorder usually experience muscle weakness (myopathy), poor muscle tone (hypotonia), an enlarged liver (hepatomegaly), and heart defects. Affected infants might also fail to gain weight and grow at the expected charge (failure to thrive) and have respiration problems. If untreated, this type of Pompe disease results in dying from coronary heart failure in the primary year of life. The non-basic form of infantile-onset Pompe disease usually seems by age 1. It is characterized by delayed motor abilities (reminiscent of rolling over and sitting) and progressive muscle weakness.
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Ivy, John & Portman, Robert. Nutrient Timing: The way forward for Sports Nutrition. Aragon, Alan Albert; Schoenfeld, Brad Jon (2013-01-29). "Nutrient timing revisited: is there a put up-train anabolic window?". Journal of the International Society of Sports Nutrition. Stark, Matthew; Lukaszuk, Judith; Prawitz, Aimee; Salacinski, Amanda (2012-12-14). "Protein timing and its results on muscular hypertrophy and power in individuals engaged in weight-training". Journal of the International Society of Sports Nutrition. Witard, Oliver C.; Jackman, Sarah R.; Breen, Leigh; Smith, Kenneth; Selby, Anna; Tipton, Kevin D. (January 2014). "Myofibrillar muscle protein synthesis rates subsequent to a meal in response to rising doses of whey protein at rest and after resistance train". The American Journal of Clinical Nutrition. Murray, Bob; Rosenbloom, Christine (2018-04-01).